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Clark, M R
Shohet, S B
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Clark, M R
Shohet, S B
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Clark, M R
Shohet, S B
spellingShingle Galili, U
Clark, M R
Shohet, S B
Journal of Clinical Investigation
Excessive binding of natural anti-alpha-galactosyl immunoglobin G to sickle erythrocytes may contribute to extravascular cell destruction.
General Medicine
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title Excessive binding of natural anti-alpha-galactosyl immunoglobin G to sickle erythrocytes may contribute to extravascular cell destruction.
title_unstemmed Excessive binding of natural anti-alpha-galactosyl immunoglobin G to sickle erythrocytes may contribute to extravascular cell destruction.
title_full Excessive binding of natural anti-alpha-galactosyl immunoglobin G to sickle erythrocytes may contribute to extravascular cell destruction.
title_fullStr Excessive binding of natural anti-alpha-galactosyl immunoglobin G to sickle erythrocytes may contribute to extravascular cell destruction.
title_full_unstemmed Excessive binding of natural anti-alpha-galactosyl immunoglobin G to sickle erythrocytes may contribute to extravascular cell destruction.
title_short Excessive binding of natural anti-alpha-galactosyl immunoglobin G to sickle erythrocytes may contribute to extravascular cell destruction.
title_sort excessive binding of natural anti-alpha-galactosyl immunoglobin g to sickle erythrocytes may contribute to extravascular cell destruction.
topic General Medicine
url http://dx.doi.org/10.1172/jci112286
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spelling Galili, U Clark, M R Shohet, S B 0021-9738 American Society for Clinical Investigation General Medicine http://dx.doi.org/10.1172/jci112286 Excessive binding of natural anti-alpha-galactosyl immunoglobin G to sickle erythrocytes may contribute to extravascular cell destruction. Journal of Clinical Investigation
spellingShingle Galili, U, Clark, M R, Shohet, S B, Journal of Clinical Investigation, Excessive binding of natural anti-alpha-galactosyl immunoglobin G to sickle erythrocytes may contribute to extravascular cell destruction., General Medicine
title Excessive binding of natural anti-alpha-galactosyl immunoglobin G to sickle erythrocytes may contribute to extravascular cell destruction.
title_full Excessive binding of natural anti-alpha-galactosyl immunoglobin G to sickle erythrocytes may contribute to extravascular cell destruction.
title_fullStr Excessive binding of natural anti-alpha-galactosyl immunoglobin G to sickle erythrocytes may contribute to extravascular cell destruction.
title_full_unstemmed Excessive binding of natural anti-alpha-galactosyl immunoglobin G to sickle erythrocytes may contribute to extravascular cell destruction.
title_short Excessive binding of natural anti-alpha-galactosyl immunoglobin G to sickle erythrocytes may contribute to extravascular cell destruction.
title_sort excessive binding of natural anti-alpha-galactosyl immunoglobin g to sickle erythrocytes may contribute to extravascular cell destruction.
title_unstemmed Excessive binding of natural anti-alpha-galactosyl immunoglobin G to sickle erythrocytes may contribute to extravascular cell destruction.
topic General Medicine
url http://dx.doi.org/10.1172/jci112286