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Excessive binding of natural anti-alpha-galactosyl immunoglobin G to sickle erythrocytes may contribute to extravascular cell destruction.
Gespeichert in:
Zeitschriftentitel: | Journal of Clinical Investigation |
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Personen und Körperschaften: | , , |
In: | Journal of Clinical Investigation, 77, 1986, 1, S. 27-33 |
Format: | E-Article |
Sprache: | Englisch |
veröffentlicht: |
American Society for Clinical Investigation
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Schlagwörter: |
author_facet |
Galili, U Clark, M R Shohet, S B Galili, U Clark, M R Shohet, S B |
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author |
Galili, U Clark, M R Shohet, S B |
spellingShingle |
Galili, U Clark, M R Shohet, S B Journal of Clinical Investigation Excessive binding of natural anti-alpha-galactosyl immunoglobin G to sickle erythrocytes may contribute to extravascular cell destruction. General Medicine |
author_sort |
galili, u |
spelling |
Galili, U Clark, M R Shohet, S B 0021-9738 American Society for Clinical Investigation General Medicine http://dx.doi.org/10.1172/jci112286 Excessive binding of natural anti-alpha-galactosyl immunoglobin G to sickle erythrocytes may contribute to extravascular cell destruction. Journal of Clinical Investigation |
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American Society for Clinical Investigation, 1986 |
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1986 |
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American Society for Clinical Investigation |
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Journal of Clinical Investigation |
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title |
Excessive binding of natural anti-alpha-galactosyl immunoglobin G to sickle erythrocytes may contribute to extravascular cell destruction. |
title_unstemmed |
Excessive binding of natural anti-alpha-galactosyl immunoglobin G to sickle erythrocytes may contribute to extravascular cell destruction. |
title_full |
Excessive binding of natural anti-alpha-galactosyl immunoglobin G to sickle erythrocytes may contribute to extravascular cell destruction. |
title_fullStr |
Excessive binding of natural anti-alpha-galactosyl immunoglobin G to sickle erythrocytes may contribute to extravascular cell destruction. |
title_full_unstemmed |
Excessive binding of natural anti-alpha-galactosyl immunoglobin G to sickle erythrocytes may contribute to extravascular cell destruction. |
title_short |
Excessive binding of natural anti-alpha-galactosyl immunoglobin G to sickle erythrocytes may contribute to extravascular cell destruction. |
title_sort |
excessive binding of natural anti-alpha-galactosyl immunoglobin g to sickle erythrocytes may contribute to extravascular cell destruction. |
topic |
General Medicine |
url |
http://dx.doi.org/10.1172/jci112286 |
publishDate |
1986 |
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author | Galili, U, Clark, M R, Shohet, S B |
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spelling | Galili, U Clark, M R Shohet, S B 0021-9738 American Society for Clinical Investigation General Medicine http://dx.doi.org/10.1172/jci112286 Excessive binding of natural anti-alpha-galactosyl immunoglobin G to sickle erythrocytes may contribute to extravascular cell destruction. Journal of Clinical Investigation |
spellingShingle | Galili, U, Clark, M R, Shohet, S B, Journal of Clinical Investigation, Excessive binding of natural anti-alpha-galactosyl immunoglobin G to sickle erythrocytes may contribute to extravascular cell destruction., General Medicine |
title | Excessive binding of natural anti-alpha-galactosyl immunoglobin G to sickle erythrocytes may contribute to extravascular cell destruction. |
title_full | Excessive binding of natural anti-alpha-galactosyl immunoglobin G to sickle erythrocytes may contribute to extravascular cell destruction. |
title_fullStr | Excessive binding of natural anti-alpha-galactosyl immunoglobin G to sickle erythrocytes may contribute to extravascular cell destruction. |
title_full_unstemmed | Excessive binding of natural anti-alpha-galactosyl immunoglobin G to sickle erythrocytes may contribute to extravascular cell destruction. |
title_short | Excessive binding of natural anti-alpha-galactosyl immunoglobin G to sickle erythrocytes may contribute to extravascular cell destruction. |
title_sort | excessive binding of natural anti-alpha-galactosyl immunoglobin g to sickle erythrocytes may contribute to extravascular cell destruction. |
title_unstemmed | Excessive binding of natural anti-alpha-galactosyl immunoglobin G to sickle erythrocytes may contribute to extravascular cell destruction. |
topic | General Medicine |
url | http://dx.doi.org/10.1172/jci112286 |